96 Food Sensitvity Test
SKU:
$259.00
$259.00 - $429.00
per item
Most HSA and flex spending accounts consider this an approved expense!
Food sensitivities are recognized to be correlated with many chronic health conditions, such as IBS, eczema, fatigue, and others - but with proper identification and elimination of offending foods, many experience relief from their symptoms. The IgG, IgG4, and IgA antibody assessment panels can serve as useful tools in the identification of trigger foods in individuals.
IgG:
IgG reactions to foods are known as food sensitivities and are different from food allergies. IgG reactions trigger inflammation and are more difficult to pinpoint because reactions occur hours or even days after eating the offending food. Common symptoms include headaches, eczema, fatigue, weight gain, depression, joint pain, GI symptoms, and so much more. Not many people realize that they could feel much better than they do, just by eliminating foods that their body has a difficult time processing.
IgA:
We look at IgA in addition to IgG, to see both sides of the gut (immune system). IgG is in the blood, IgA is in the mucous membranes. IgA is the second most abundant primary immunoglobulin antibody behind IgG; they complete each other’s protective mechanisms. If there are any known mucous membrane issues, IgA should almost certainly be performed because IgA antibodies serve as a barrier between your body and the outside world.
IgG4:
IgG reactions may or may not include IgE reactions. Glycine-rich cell wall proteins may stimulate IgG responses without stimulating IgE. Human studies indicate that glycine-rich cell wall proteins may represent a missing link between food antigen exposures and autoimmune responses, and IgG4 may play a role. IgG4 is considered a blocking antibody for IgE, and in many cases that is exactly what happens. Levels of IgG4 antibodies increase in people with IgE inhalant allergies.
However, under certain circumstances IgG4 may directly stimulate complement and promote autoimmune responses, resulting in a variety of conditions including:
• Autoimmune pancreatitis
• Kidney problems
• Prostatitis
• Lymph node inflammation
• Inflammation of the aorta
• Eosinophilic esophagitis (EOE) and other problems.
These IgG4 autoimmune conditions are antigen-driven, this means that antigen avoidance may decrease symptoms or slow progression in these under-diagnosed disorders.
Food sensitivities are recognized to be correlated with many chronic health conditions, such as IBS, eczema, fatigue, and others - but with proper identification and elimination of offending foods, many experience relief from their symptoms. The IgG, IgG4, and IgA antibody assessment panels can serve as useful tools in the identification of trigger foods in individuals.
IgG:
IgG reactions to foods are known as food sensitivities and are different from food allergies. IgG reactions trigger inflammation and are more difficult to pinpoint because reactions occur hours or even days after eating the offending food. Common symptoms include headaches, eczema, fatigue, weight gain, depression, joint pain, GI symptoms, and so much more. Not many people realize that they could feel much better than they do, just by eliminating foods that their body has a difficult time processing.
IgA:
We look at IgA in addition to IgG, to see both sides of the gut (immune system). IgG is in the blood, IgA is in the mucous membranes. IgA is the second most abundant primary immunoglobulin antibody behind IgG; they complete each other’s protective mechanisms. If there are any known mucous membrane issues, IgA should almost certainly be performed because IgA antibodies serve as a barrier between your body and the outside world.
IgG4:
IgG reactions may or may not include IgE reactions. Glycine-rich cell wall proteins may stimulate IgG responses without stimulating IgE. Human studies indicate that glycine-rich cell wall proteins may represent a missing link between food antigen exposures and autoimmune responses, and IgG4 may play a role. IgG4 is considered a blocking antibody for IgE, and in many cases that is exactly what happens. Levels of IgG4 antibodies increase in people with IgE inhalant allergies.
However, under certain circumstances IgG4 may directly stimulate complement and promote autoimmune responses, resulting in a variety of conditions including:
• Autoimmune pancreatitis
• Kidney problems
• Prostatitis
• Lymph node inflammation
• Inflammation of the aorta
• Eosinophilic esophagitis (EOE) and other problems.
These IgG4 autoimmune conditions are antigen-driven, this means that antigen avoidance may decrease symptoms or slow progression in these under-diagnosed disorders.